Hess, K; Alzahrani, S H; Mathai, M; Schroeder, V; Carter, A M; Howell, G; Koko, T; Strachan, M W J; Price, J F; Smith, K A; Grant, P J; Ajjan, R A (2012). A novel mechanism for hypofibrinolysis in diabetes: the role of complement C3. Diabetologia, 55(4), pp. 1103-1113. Heidelberg: Springer 10.1007/s00125-011-2301-7
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Hess2012_Article_ANovelMechanismForHypofibrinol.pdf - Published Version Available under License Publisher holds Copyright. Download (752kB) | Preview |
Impaired fibrin clot lysis is a key abnormality in diabetes and complement C3 is one protein identified in blood clots. This work investigates the mechanistic pathways linking C3 and hypofibrinolysis in diabetes using ex vivo/in vitro studies.
Item Type: |
Journal Article (Original Article) |
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Division/Institute: |
04 Faculty of Medicine > Department of Haematology, Oncology, Infectious Diseases, Laboratory Medicine and Hospital Pharmacy (DOLS) > Clinic of Haematology and Central Haematological Laboratory |
UniBE Contributor: |
Schröder, Verena |
ISSN: |
0012-186X |
Publisher: |
Springer |
Language: |
English |
Submitter: |
Factscience Import |
Date Deposited: |
04 Oct 2013 14:35 |
Last Modified: |
05 Dec 2022 14:11 |
Publisher DOI: |
10.1007/s00125-011-2301-7 |
PubMed ID: |
21918806 |
Web of Science ID: |
000301182000026 |
BORIS DOI: |
10.48350/13829 |
URI: |
https://boris.unibe.ch/id/eprint/13829 (FactScience: 220495) |
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