DNA repair, gap suppression, or fork protection: BRCA2 needs a break!

Dibitetto, Diego; Rottenberg, Sven (2024). DNA repair, gap suppression, or fork protection: BRCA2 needs a break! Molecular cell, 84(3), pp. 409-410. Cell Press 10.1016/j.molcel.2023.12.026

[img] Text
1-s2.0-S1097276523010729-main.pdf - Published Version
Restricted to registered users only
Available under License Publisher holds Copyright.

Download (1MB) | Request a copy

In this issue of Molecular Cell, Lim et al.1 reveal new insights into the distinct roles of BRCA2 in coping with DNA breaks, highlighting homologous recombination as the pivotal function that affects tumorigenesis and therapy response.

Item Type:

Journal Article (Original Article)

Division/Institute:

05 Veterinary Medicine > Department of Infectious Diseases and Pathobiology (DIP) > Institute of Animal Pathology
05 Veterinary Medicine > Department of Infectious Diseases and Pathobiology (DIP)

UniBE Contributor:

Dibitetto, Diego, Rottenberg, Sven

Subjects:

600 Technology > 630 Agriculture

ISSN:

1097-2765

Publisher:

Cell Press

Language:

English

Submitter:

Pubmed Import

Date Deposited:

05 Feb 2024 11:46

Last Modified:

05 Feb 2024 19:12

Publisher DOI:

10.1016/j.molcel.2023.12.026

PubMed ID:

38307000

BORIS DOI:

10.48350/192418

URI:

https://boris.unibe.ch/id/eprint/192418

Actions (login required)

Edit item Edit item
Provide Feedback