Reactive oxygen species mediate functional differences in human radial and internal thoracic arteries from smokers

Müller-Schweinitzer, Else; Müller, Sandra E; Reineke, David C; Kern, Thomas; Carrel, Thierry P; Eckstein, Friedrich S; Grapow, Martin T R (2010). Reactive oxygen species mediate functional differences in human radial and internal thoracic arteries from smokers. Journal of vascular surgery, 51(2), pp. 438-44. Amsterdam: Elsevier 10.1016/j.jvs.2009.09.040

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Smoking not only increases the risk that coronary heart disease will develop but also morbidity and mortality in patients with known coronary atherosclerosis and after coronary artery bypass grafting. Excessive generation of reactive oxygen species (ROS) has been implicated as the final common pathway for the development of endothelial dysfunction in various cardiovascular risk factors. This study assessed the influence of smoking on two different human arteries routinely used as coronary artery bypass graft conduits.

Item Type:

Journal Article (Original Article)

Division/Institute:

04 Faculty of Medicine > Department of Cardiovascular Disorders (DHGE) > Clinic of Cardiovascular Surgery

UniBE Contributor:

Müller, Simon Andreas; Reineke, David Christian and Carrel, Thierry

ISSN:

0741-5214

Publisher:

Elsevier

Language:

English

Submitter:

Factscience Import

Date Deposited:

04 Oct 2013 14:13

Last Modified:

17 Mar 2015 19:15

Publisher DOI:

10.1016/j.jvs.2009.09.040

PubMed ID:

20036100

Web of Science ID:

000274602800021

URI:

https://boris.unibe.ch/id/eprint/2745 (FactScience: 205618)

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