The lectin-like domain of tumor necrosis factor improves lung function after rat lung transplantation--potential role for a reduction in reactive oxygen species generation

Hamacher, Jürg; Stammberger, Uz; Roux, Jeremie; Kumar, Sanjiv; Yang, Guang; Xiong, Chenling; Schmid, Ralph A; Fakin, Richard M; Chakraborty, Trinad; Hossain, Hamid M D; Pittet, Jean-François; Wendel, Albrecht; Black, Stephen M; Lucas, Rudolf (2010). The lectin-like domain of tumor necrosis factor improves lung function after rat lung transplantation--potential role for a reduction in reactive oxygen species generation. Critical care medicine, 38(3), pp. 871-8. Baltimore, Md.: Lippincott Williams & Wilkins 10.1097/CCM.0b013e3181cdf725

Full text not available from this repository.

To test the hypothesis that the lectin-like domain of tumor necrosis factor, mimicked by the TIP peptide, can improve lung function after unilateral orthotopic lung isotransplantation. Because of a lack of a specific treatment for ischemia reperfusion-mediated lung injury, accompanied by a disrupted barrier integrity and a dysfunctional alveolar liquid clearance, alternative therapies restoring these parameters after lung transplantation are required.

Item Type:	Journal Article (Original Article)
Division/Institute:	04 Faculty of Medicine > Department of Gastro-intestinal, Liver and Lung Disorders (DMLL) > Clinic of Thoracic Surgery
UniBE Contributor:	Schmid, Ralph
ISSN:	0090-3493
Publisher:	Lippincott Williams & Wilkins
Language:	English
Submitter:	Factscience Import
Date Deposited:	04 Oct 2013 14:07
Last Modified:	05 Dec 2022 14:00
Publisher DOI:	10.1097/CCM.0b013e3181cdf725
PubMed ID:	20081530
Web of Science ID:	000275266200019
URI:	https://boris.unibe.ch/id/eprint/282 (FactScience: 197301)