Na+ channel function, regulation, structure, trafficking and sequestration.

Chen-Izu, Ye; Shaw, Robin M; Pitt, Geoffrey S; Yarov-Yarovoy, Vladimir; Sack, Jon T; Abriel, Hugues; Aldrich, Richard W; Belardinelli, Luiz; Cannell, Mark B; Catterall, William A; Chazin, Walter J; Chiamvimonvat, Nipavan; Deschenes, Isabelle; Grandi, Eleonora; Hund, Thomas J; Izu, Leighton T; Maier, Lars S; Maltsev, Victor A; Marionneau, Celine; Mohler, Peter J; ... (2015). Na+ channel function, regulation, structure, trafficking and sequestration. Journal of physiology, 593(6), pp. 1347-1360. Wiley-Blackwell 10.1113/jphysiol.2014.281428

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This paper is the second of a series of three reviews published in this issue resulting from the University of California Davis Cardiovascular Symposium 2014: Systems approach to understanding cardiac excitation-contraction coupling and arrhythmias: Na(+) channel and Na(+) transport. The goal of the symposium was to bring together experts in the field to discuss points of consensus and controversy on the topic of sodium in the heart. The present review focuses on Na(+) channel function and regulation, Na(+) channel structure and function, and Na(+) channel trafficking, sequestration and complexing.

Item Type:

Journal Article (Original Article)

Division/Institute:

04 Faculty of Medicine > Pre-clinic Human Medicine > BioMedical Research (DBMR) > DBMR Forschung Mu35 > Forschungsgruppe Ionenkanalkrankheiten
04 Faculty of Medicine > Pre-clinic Human Medicine > BioMedical Research (DBMR) > DBMR Forschung Mu35 > Forschungsgruppe Ionenkanalkrankheiten

UniBE Contributor:

Abriel, Hugues

Subjects:

600 Technology > 610 Medicine & health

ISSN:

0022-3751

Publisher:

Wiley-Blackwell

Language:

English

Submitter:

Verena de Serra Frazao-Bill

Date Deposited:

13 Apr 2016 08:08

Last Modified:

07 Nov 2016 09:36

Publisher DOI:

10.1113/jphysiol.2014.281428

PubMed ID:

25772290

BORIS DOI:

10.7892/boris.80629

URI:

https://boris.unibe.ch/id/eprint/80629

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