IVIG blocks complement deposition mediated by anti-GM1 antibodies in multifocal motor neuropathy

Yuki, N; Watanabe, H; Nakajima, T; Späth, P J (2011). IVIG blocks complement deposition mediated by anti-GM1 antibodies in multifocal motor neuropathy. Journal of neurology, neurosurgery and psychiatry, 82(1), pp. 87-91. London: BMJ Publishing Group 10.1136/jnnp.2010.205856

Full text not available from this repository. (Request a copy)

The pathogenesis of multifocal motor neuropathy (MMN) has yet to be established. MMN patients often carry anti-GM1 IgM antibodies, suggesting an autoimmune process involving complement. Intravenous immunoglobulin (IVIG) is the first line treatment, but its action mechanism is unknown.

Item Type:

Journal Article (Original Article)

Division/Institute:

04 Faculty of Medicine > Pre-clinic Human Medicine > Institute of Pharmacology

UniBE Contributor:

Späth, Peter Julius

ISSN:

0022-3050

Publisher:

BMJ Publishing Group

Language:

English

Submitter:

Factscience Import

Date Deposited:

04 Oct 2013 14:09

Last Modified:

06 Dec 2013 13:20

Publisher DOI:

10.1136/jnnp.2010.205856

PubMed ID:

20667861

Web of Science ID:

000285183000021

URI:

https://boris.unibe.ch/id/eprint/901 (FactScience: 201215)

Actions (login required)

Edit item Edit item
Provide Feedback