Gassmann, Max

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Journal Article

Vavricka, Stephan; Ruiz, Pedro A; Scharl, Sylvie; Biedermann, Luc; Scharl, Michael; de Vallière, Cheryl; Lundby, Carsten; Wenger, Roland H; Held, Leonhard; Merz, Tobias; Gassmann, Max; Lutz, Thomas; Kunz, Andres; Bron, Denis; Fontana, Adriano; Strauss, Laura; Weber, Achim; Fried, Michael; Rogler, Gerhard and Zeitz, Jonas (2017). Protocol for a prospective, controlled, observational study to evaluate the influence of hypoxia on healthy volunteers and patients with inflammatory bowel disease: the Altitude IBD Study. BMJ open, 7(1), e013477. BMJ Publishing Group 10.1136/bmjopen-2016-013477

Balmer, Jasmin; Ji, Rui; Ray, Thomas A.; Selber, Fabia; Gassmann, Max; Peachey, Neal S.; Gregg, Ronald G.; Enzmann, Volker (2013). Presence of the Gpr179(nob5) allele in a C3H-derived transgenic mouse. Molecular vision, 19, pp. 2615-2625. Emory University

Richter, Vincent; Savery, Michele D; Gassmann, Max; Baum, Oliver; Damiano, Edward R; Pries, Axel R (2011). Excessive erythrocytosis compromises the blood-endothelium interface in erythropoietin-overexpressing mice. Journal of physiology, 589(Pt 21), pp. 5181-92. Oxford: Wiley-Blackwell 10.1113/jphysiol.2011.209262

Heinicke, Katja; Baum, Oliver; Ogunshola, Omolara O; Vogel, Johannes; Stallmach, Thomas; Wolfer, David P; Keller, Stephan; Weber, Klaus; Wagner, Peter D; Gassmann, Max; Djonov, Valentin (2006). Excessive erythrocytosis in adult mice overexpressing erythropoietin leads to hepatic, renal, neuronal, and muscular degeneration. American journal of physiology - regulatory, integrative and comparative physiology, 291(4), R947-56. Bethesda, Md.: American Physiological Society 10.1152/ajpregu.00152.2006

Däpp, Christoph; Gassmann, Max; Hoppeler, Hans; Flück, Martin (2006). Hypoxia-induced gene activity in disused oxidative muscle. Advances in experimental medicine and biology, 588, pp. 171-188. Wien: Springer

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