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Althaus, Christian L.; Joos, Beda; Perelson, Alan S.; Günthard, Huldrych F. (2014). Quantifying the turnover of transcriptional subclasses of HIV-1-infected cells. PLoS computational biology, 10(10), e1003871. Public Library of Science 10.1371/journal.pcbi.1003871
Di Giallonardo, Francesca; Zagordi, Osvaldo; Duport, Yannick; Leemann, Christine; Joos, Beda; Künzli-Gontarczyk, Marzanna; Bruggmann, Rémy; Beerenwinkel, Niko; Günthard, Huldrych F.; Metzner, Karin J. (2013). Next-generation sequencing of HIV-1 RNA genomes: determination of error rates and minimizing artificial recombination. PLoS ONE, 8(9), e74249. Public Library of Science 10.1371/journal.pone.0074249
Wyl, Viktor von; Gianella, Sara; Fischer, Marek; Niederoest, Barbara; Kuster, Herbert; Battegay, Manuel; Bernasconi, Enos; Cavassini, Matthias; Rauch, Andri; Hirschel, Bernard; Vernazza, Pietro; Weber, Rainer; Joos, Beda; Günthard, Huldrych F; Swiss HIV Cohort Study-SHCS, (2011). Early antiretroviral therapy during primary HIV-1 infection results in a transient reduction of the viral setpoint upon treatment interruption. PLoS ONE, 6(11), e27463. Lawrence, Kans.: Public Library of Science 10.1371/journal.pone.0027463
Gianella, Sara; von Wyl, Viktor; Fischer, Marek; Niederoest, Barbara; Battegay, Manuel; Bernasconi, Enos; Cavassini, Matthias; Rauch, Andri; Hirschel, Bernard; Vernazza, Pietro; Weber, Rainer; Joos, Beda; Günthard, Huldrych F; Swiss HIV Cohort Study, (2011). Effect of early antiretroviral therapy during primary HIV-1 infection on cell-associated HIV-1 DNA and plasma HIV-1 RNA. Antiviral therapy, 16(4), pp. 535-45. London: International Medical Press 10.3851/IMP1776
Kouyos, Roger D; von Wyl, Viktor; Yerly, Sabine; Böni, Jürg; Rieder, Philip; Joos, Beda; Taffé, Patrick; Shah, Cyril; Bürgisser, Philippe; Klimkait, Thomas; Weber, Rainer; Hirschel, Bernard; Cavassini, Matthias; Rauch, Andri; Battegay, Manuel; Vernazza, Pietro L; Bernasconi, Enos; Ledergerber, Bruno; Bonhoeffer, Sebastian; Günthard, Huldrych F; ... (2011). Ambiguous nucleotide calls from population-based sequencing of HIV-1 are a marker for viral diversity and the age of infection. Clinical infectious diseases, 52(4), pp. 532-9. Cary, N.C.: Oxford University Press 10.1093/cid/ciq164
