Ullrich, N D

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Number of items: 9.

Journal Article

Gonzales, C; Ullrich, N D; Gerber, S; Berthonneche, C; Niggli, E; Pedrazzini, T (2012). Isolation of cardiovascular precursor cells from the human fetal heart. Tissue engineering, 18(1-2), pp. 198-207. Larchmont, N.Y.: Mary Ann Liebert 10.1089/ten.tea.2011.0022

Ullrich, N D; Valdivia, H H; Niggli, E (2012). phosphorylation of cardiac ryanodine receptor modulates SR luminal Ca2+ sensitivity. Journal of molecular and cellular cardiology, 53(1), pp. 33-42. Oxford: Elsevier 10.1016/j.yjmcc.2012.03.015

Kyrychenko, S; Polàkova, E; Ullrich, N D; Niggli, E; Shirokova, N (2012). Insights into RyRs dysfunctions via studies of intracellular calcium signals. Biophysical journal, 102, 213a. Bethesda, Md.: Biophysical Society

Conference or Workshop Item

Marcu, I C; Hoffmann, P; Jaconi, M; Ullrich, N D (2012). Functional characterization of stem cell-derived cardiomyocytes. In: Conference Proceedings of ICGEB/CVR Symposium.

Martins, A; Shkyl, V M; Ullrich, N D; Nowycky, M; Niggli, E; Shirokova, N (2008). Reciprocal amplification of ROS and Ca2+ signals in stressed MDX dystrophic skeletal muscle fibres. In: 7th Swiss Meeting on Muscle Research, Magglingen, Switzerland. 2008.

Ullrich, N D; Fanchaouy, J; Gusev, K; Polakova, E; Shirokova, N; Niggli, E (2008). Changes of EC-coupling and RyR Ca2+ sensitivity in dystrophic mdx mouse cardiomyocytes. Poster. In: Day of Clinical Research, University Hospital, Bern. 2008.

Ullrich, N D; Egger, M (2008). Cardiac IP3 receptor function and communication with RyRs in mouse atrial myocytes. In: Poster at the SCRTN meeting, 2008.

Ullrich, N D; Fanchaouy, M; Gusev, K; Polakova, E; Shirokova, N; Niggli, E (2008). Changes of EC-coupling and RyR Ca2+ sensitivity in dystrophic mdx mouse cardiomyocytes. In: Annual meeting of the Swiss Cardiovascular Research and Training Network, Bern. Eur J Pharm Biopharm 2008.

Ullrich, N D; Fanchaouy, M; Gusev, K; Polakova, E; Shirokova, N; Niggli, E (2008). Disruption of the cytoskeletal dystrophin complex alters excitation-contraction coupling mechanisms in cardiomyocytes of the MDX mouse. In: 7th Swiss Meeting on Muscle Research, Magglingen, Switzerland. 2008.

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