Raake, Philip W; Zhang, Xiaoying; Vinge, Leif E; Brinks, Henriette; Gao, Erhe; Jaleel, Naser; Li, Yingxin; Tang, Mingxin; Most, Patrick; Dorn, Gerald W; Houser, Steven R; Katus, Hugo A; Chen, Xiongwen; Koch, Walter J (2012). Cardiac G-protein-coupled receptor kinase 2 ablation induces a novel Ca2+ handling phenotype resistant to adverse alterations and remodeling after myocardial infarction. Circulation, 125(17), pp. 2108-18. Baltimore, Md.: Lippincott Williams & Wilkins 10.1161/CIRCULATIONAHA.111.044255
Full text not available from this repository. (Request a copy)G-protein-coupled receptor kinase 2 (GRK2) is a primary regulator of β-adrenergic signaling in the heart. G-protein-coupled receptor kinase 2 ablation impedes heart failure development, but elucidation of the cellular mechanisms has not been achieved, and such elucidation is the aim of this study.
Item Type: |
Journal Article (Original Article) |
|---|---|
Division/Institute: |
04 Faculty of Medicine > Department of Cardiovascular Disorders (DHGE) > Clinic of Heart Surgery |
UniBE Contributor: |
Most, Henriette |
Subjects: |
600 Technology > 610 Medicine & health |
ISSN: |
0009-7322 |
Publisher: |
Lippincott Williams & Wilkins |
Language: |
English |
Submitter: |
Factscience Import |
Date Deposited: |
04 Oct 2013 14:35 |
Last Modified: |
27 Feb 2024 14:29 |
Publisher DOI: |
10.1161/CIRCULATIONAHA.111.044255 |
PubMed ID: |
22496128 |
Web of Science ID: |
000306959200020 |
URI: |
https://boris.unibe.ch/id/eprint/14195 (FactScience: 221050) |
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