Rimoldi, Stefano F; Yuzefpolskaya, Melana; Allemann, Yves; Messerli, Franz (2009). Flash pulmonary edema. Progress in cardiovascular diseases, 52(3), pp. 249-59. Orlando, Fla.: Elsevier 10.1016/j.pcad.2009.10.002
Full text not available from this repository.Flash pulmonary edema (FPE) is a general clinical term used to describe a particularly dramatic form of acute decompensated heart failure. Well-established risk factors for heart failure such as hypertension, coronary ischemia, valvular heart disease, and diastolic dysfunction are associated with acute decompensated heart failure as well as with FPE. However, endothelial dysfunction possibly secondary to an excessive activity of renin-angiotensin-aldosterone system, impaired nitric oxide synthesis, increased endothelin levels, and/or excessive circulating catecholamines may cause excessive pulmonary capillary permeability and facilitate FPE formation. Renal artery stenosis particularly when bilateral has been identified has a common cause of FPE. Lack of diurnal variation in blood pressure and a widened pulse pressure have been identified as risk factors for FPE. This review is an attempt to delineate clinical and pathophysiological mechanisms responsible for FPE and to distinguish pathophysiologic, clinical, and therapeutic aspects of FPE from those of acute decompensated heart failure.
Item Type: |
Journal Article (Further Contribution) |
|---|---|
Division/Institute: |
04 Faculty of Medicine > Department of Cardiovascular Disorders (DHGE) > Clinic of Cardiology |
UniBE Contributor: |
Rimoldi, Stefano, Allemann, Yves |
ISSN: |
0033-0620 |
ISBN: |
19917337 |
Publisher: |
Elsevier |
Language: |
English |
Submitter: |
Factscience Import |
Date Deposited: |
04 Oct 2013 15:09 |
Last Modified: |
05 Dec 2022 14:21 |
Publisher DOI: |
10.1016/j.pcad.2009.10.002 |
PubMed ID: |
19917337 |
Web of Science ID: |
000272421800010 |
URI: |
https://boris.unibe.ch/id/eprint/30209 (FactScience: 191444) |
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