Stolp, Bettina; Fackler, Oliver T (2011). How HIV takes advantage of the cytoskeleton in entry and replication. Viruses, 3(4), pp. 293-311. Basel: Molecular Diversity Preservation International MDPI 10.3390/v3040293
Full text not available from this repository. (Request a copy)The host cell cytoskeleton plays a key role in the life cycle of viral pathogens whose propagation depends on mandatory intracellular steps. Accordingly, also the human immunodeficiency virus type 1 (HIV-1) has evolved strategies to exploit and modulate in particular the actin cytoskeleton for its purposes. This review will recapitulate recent findings on how HIV-1 hijacks the cytoskeleton to facilitate entry into, transport within and egress from host cells as well as to commandeer communication of infected with uninfected bystander cells.
Item Type: |
Journal Article (Further Contribution) |
|---|---|
Division/Institute: |
04 Faculty of Medicine > Pre-clinic Human Medicine > Theodor Kocher Institute |
UniBE Contributor: |
Stolp, Bettina |
ISSN: |
1999-4915 |
Publisher: |
Molecular Diversity Preservation International MDPI |
Language: |
English |
Submitter: |
Factscience Import |
Date Deposited: |
04 Oct 2013 14:23 |
Last Modified: |
05 Dec 2022 14:06 |
Publisher DOI: |
10.3390/v3040293 |
PubMed ID: |
21994733 |
Web of Science ID: |
000290309100002 |
URI: |
https://boris.unibe.ch/id/eprint/7813 (FactScience: 213154) |
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