ATG12 deficiency leads to tumor cell oncosis owing to diminished mitochondrial biogenesis and reduced cellular bioenergetics

Liu, He; He, Zhaoyue; Germič, Nina; Ademi, Hyrijie; Frangež, Živa; Felser, Andrea; Peng, Shuang; Riether, Carsten; Djonov, Valentin; Nuoffer, Jean-Marc; Bovet, Cédric; Mlinarič-Raščan, Irena; Zlobec, Inti; Fiedler, Georg; Perren, Aurel; Simon, Hans-Uwe (2020). ATG12 deficiency leads to tumor cell oncosis owing to diminished mitochondrial biogenesis and reduced cellular bioenergetics. Cell death and differentiation, 27(6), pp. 1965-1980. Springer Nature 10.1038/s41418-019-0476-5

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Item Type:

Journal Article (Original Article)

Division/Institute:

09 Interdisciplinary Units > Microscopy Imaging Center (MIC)
04 Faculty of Medicine > Department of Haematology, Oncology, Infectious Diseases, Laboratory Medicine and Hospital Pharmacy (DOLS) > Institute of Clinical Chemistry
04 Faculty of Medicine > Department of Gynaecology, Paediatrics and Endocrinology (DFKE) > Clinic of Paediatric Medicine
04 Faculty of Medicine > Pre-clinic Human Medicine > Institute of Anatomy
04 Faculty of Medicine > Pre-clinic Human Medicine > Institute of Pharmacology
04 Faculty of Medicine > Service Sector > Institute of Pathology
04 Faculty of Medicine > Department of Haematology, Oncology, Infectious Diseases, Laboratory Medicine and Hospital Pharmacy (DOLS) > Clinic of Medical Oncology

UniBE Contributor:

Liu, He; He, Zhaoyue; Germic, Nina; Frangez, Ziva; Felser, Andrea Debora; Peng, Shuang; Riether, Carsten; Djonov, Valentin Georgiev; Nuoffer, Jean-Marc; Bovet, Cédric; Zlobec, Inti; Fiedler, Georg Martin; Perren, Aurel and Simon, Hans-Uwe

Subjects:

600 Technology > 610 Medicine & health
500 Science > 570 Life sciences; biology

ISSN:

1350-9047

Publisher:

Springer Nature

Language:

English

Submitter:

Alexandra Müller

Date Deposited:

06 Jan 2020 10:38

Last Modified:

25 May 2020 01:31

Publisher DOI:

10.1038/s41418-019-0476-5

PubMed ID:

31844253

Additional Information:

These authors contributed equally: He Liu, Zhaoyue He

BORIS DOI:

10.7892/boris.137005

URI:

https://boris.unibe.ch/id/eprint/137005

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